Obesity has
become a huge problem in the world’s population with about 13% (about 650
million) of the adult population being obese in 2016. Most people attribute
obesity to eating too much food and unhealthy food and not exercising enough.
New research has come to light that indicates that obesity may be caused from
genetics. Variants in a gene called Ankyrin-B has become a new focus for
obesity studies. In a study using mice, it was found that variations in this
gene causes fat cells to intake glucose at a faster rate than normal and in
people with an aging metabolism or high-fat diet, it is near impossible not to
become obese.
Researchers
have been able to link variations in Ankyrin-B genes to diseases including
autism, muscular dystrophy, aging, diabetes, and irregular heartbeats. Studies
were done by knocking out and creating different mutants of Ankyrin-B gene in
mice. It was found that mice with these mutant genes became obese while having
the same diet and exercise as the other mice with normal Ankyrin-B genes. The
results showed the excess amount of lipids in the fat cells leaked into the
liver and muscles which led to inflammation and disruption of response in
insulin. It was found that mutating the Ankyrin-B genes led to the change of
Glut4 which allows glucose to enter the fat cells at a faster rate.
Observational
research needs to be done with the general population looking at whether or not
obese people have these mutated Ankyrin-B genes as well as looking at familial
histories, height, weight and glucose metabolism in order to determine the
effect of these variants on the human population.
“New Research Shows How a Gene Can Fuel Obesity in Mice.” News-Medical.net, 16 Nov. 2017, www.news-medical.net/news/20171116/New-research-shows-how-a-gene-can-fuel-obesity-in-mice.aspx.
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